The Muscle and Movement lecture presented the scientific basis for synthesizing human movement in an evolutionary context. Furthermore, it also explained muscle control in terms of fine control of force and movements. As the lecture explained, muscle drives movement and serves as a link between performance and evolution of fitness of creatures and humans alike. Muscle functions play an active role in not only stabilizing us, but also serving as a power source or motor for driving movements. Specifically, the cross-bridge cycle generates muscle force by catalyzing break down of ATP, yielding energy that is transduced to a shape change that ultimately drives mechanical processes within the body. The performance of muscle is thus driven by molecular interactions. Furthermore, the performance of muscle can be described by a force-length and force-velocity relationship. These relationships basically dictate that muscle has an optimum length for maximizing force production and that rapid shortening of the muscle reduces the ability to exert force. I tried to combine these elements of cross-bridges and Muscle Protein within my dance to show that there is a decline in force as muscles stretch and that muscles can either be strong and immobile, or fast and weak. Furthermore, to tie these processes in with prions, I tried to perform movements that illustrate the biology of prions. Specifically, I wanted to depict how the primary alpha structure of the prion is converted into a type of structure with increased beta sheet through different types of spinning, dance chain- sequences, and repetition. This conversion of alpha helices into beta sheets shows a conformational change in the prion synthesis, and within the dance movement sequence itself.